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Overall, the application of these neurons successfully models key pathological attributes that are characteristic of AD, including aggregation of amyloid beta, modified production of A40 or A42, hyperphosphorylation of TAU, reduction in synapses and their plasticity as well as amplification of oxidative stress [44]

Int J Dev Neurosci 28:127132 Al-sharefi A, Bilous R (2015) Reversible weakness and encephalopathy while on long-term valproate treatment due to carnitine deficiency

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