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T., Wang, S., Liu, H., & Yin, Y

Continuous modulation of glycine concentrations in cerebrospinal fluid (CSF) was relevant for the therapeutic efficacy

Inflammation in the CNS increases the risk of AD initiation and exacerbates the amyloid and tau pathology and the cognitive decline [12]

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The connexin43 mimetic peptide Gap19 inhibits hemichannels without altering gap junctional communication in astrocytes

While these registered trials are encouraging indicators of increasing clinical activity, none are examining specific named peptide compounds in isolation, making it impossible to draw peptide-specific conclusions from their eventual results