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High homocysteine levels, or hyperhomocysteinemia, can indicate low levels of folate and vitamin B12

It seeks to elucidate the molecular and cellular pathways through which arsenic damages renal structures, with a focus on oxidative stress, mitochondrial dysfunction, apoptosis, ferroptosis, and inflammation

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Comparative studies on binding of 3 different ligands to the N -methyl-d-aspartate recognition domain in brain synaptic membranes treated with Triton X-100

In conclusion, the gut microbiota-hepatic ferroptosis plays a crucial role in the pathogenesis of ALD, targeting the modulation of gut microbiota while inhibiting ferroptosis presents a promising therapeutic intervention to mitigate liver injury and improve outcomes for individuals affected by ALD