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Ferroptosis is an iron-dependent regulated necrosis that is considered a byproduct of cellular metabolism because of its close association with oxygen, iron, and lipid metabolism (116), it is dependent on the metabolites reactive oxygen species (ROS), phospholipids containing polyunsaturated fatty acid chains (PUFA-PL), and the transition metal iron, and exhibits biochemical features of iron accumulation and unrestricted lipid peroxidation, as well as morphologic alterations such as loss of plasma membrane integrity, mitochondrial abnormalities, cytoplasmic swelling, and moderate chromatin condensation (117, 118)

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G-Actin Sequestration and Cytoskeletal Remodelling TB-500 functions as the major G-actin sequestering molecule in cells, controlling actin polymerisation processes that drive cellular migration and tissue repair

Emerging photothermal-derived multimodal synergistic therapy in combating bacterial infections

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