ghk-cu upregulates dna repair genes

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Immunity (2018) 48(1):107119 e4

A., Chan, K

Pierce, A

Because only the liganded receptor is desensitized by this mechanism, it is called homologous desensitization the affinity for -arrestin may be increased in a ligand occupation and GRK-independent manner through phosphorylation of different ser/thr sites (but also of IL-3 and the C-terminal tail) by PKC and PKA