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Augmenting brain B 12 with fingolimod or potentially related molecules could enhance both current and future MS therapies. In their paper, the team at Sanford Burnham Prebys, with collaborators at University of Southern California, Juntendo University in Japan, Tokyo University of Pharmacy and Life Sciences and State University of New York, focused on the molecular functioning of FTY720 or fingolimod (Gilenya), a sphingosine 1-phosphate (S1P) receptor modulator that suppresses distribution of T and B immune cells errantly attacking the brains of MS patients

Full trial results and Research Summary: nej.md/4ehQwLk In the REDEFINE 2 phase 3a trial involving adults with obesity and diabetes, the use of cagrilintidesemaglutide resulted in a significantly lower body weight than placebo at 68 weeks

Schuck, T

Because growth hormone and insulin counterbalance each other, excess stimulation may lead to insulin resistance, a risk not well studied in peptide use

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