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Luteotrophic hormones such as luteinizing hormone, prolactin, and prostaglandin E2 promote corpus luteum maintenance by activating key signaling pathways, including cyclic adenosine monophosphate/protein kinase A and mitogen-activated protein kinase, while luteolytic factors such as prostaglandin F2 initiate regression through calcium- and protein kinase C-mediated pathways

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As xenobiotics build up when these three drug-metabolizing enzymes are blocked, toxicity may result (293)

Neuroendocrinology 36:130137 Demarest KT, Riegle GD, Moore KE (1984) Prolactin-induced activation of tuberoinfundibular dopaminergic neurons: evidence for both a rapid tonic and a delayed delayed component

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