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SMT, an inhibitor of iNOS [32], exerts a protective effect against erastin-induced ferroptosis in hepatocytes, and also abrogates erastin-induced accumulation of cellular NO, ROS and lipid-ROS

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Figure 6 summarizes the temporal dynamics of these processes, highlighting their overlap and their differential impact on gray and white matter vascular development

Authors' contributions MAG and QP drafted the manuscript

As mitochondria are a central hub for energy production in neurons, they are particularly vulnerable to oxidative damage