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Unraveling adipose tissue dysfunction: molecular Mechanisms, novel Biomarkers, and therapeutic targets for liver fat deposition

10.1186/ar1765 Arthritis Res

[DOI] [PMC free article] [PubMed] [Google Scholar] 201.Fu D., Yu Y., Folick A., Currie E., Farese R.V., Tsai T.-H., Xie X.S., Wang M.C

These two major pathological hallmarks contribute to changes in numerous cellular and subcellular events, such as mitochondrial dysfunction, redox imbalance, and neuroinflammation that lead to activation of neurotoxic cascade towards cell death, thereby impairing synaptic communication [81,82]

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OTUD5 counteracts UBR5-mediated K48 ubiquitylation, overcome by TRIP12 To investigate whether OTUD5 counteracted ubiquitin chain assembly, we conducted an in vitro ubiquitylation/deubiquitylation assay using wild-type or catalytically inactive (C224R) OTUD5 (OTUD5C224R)