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In summary, our data provided compelling evidence demonstrating that Tim-AIII promotes NSCLC ferroptosis by targeting and facilitating HSP90-mediated GPX4 ubiquitination and degradation, which in turn causes cell cycle arrest, inhibition of cell proliferation, and migration

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Activates the SIRT3 pathway, alleviates hepatic inflammation and oxidative stress, protects hepatocytes, and slows MASH progression

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