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In a study of colon cancer cells, COX-2 expression and PGE2 synthesis were upregulated by the IGF-2/IGF-1R autocrine pathway and IGF-1R blockade decreased COX-2 activity and inhibited tumor cell proliferation and promoted apoptosis (103)

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Therefore, reduced antioxidant capacity appears to be a consequence of disease progression and tissue damage rather than an initiating factor present throughout the entire disease course

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