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Jabado, N., Canonne-Hergaux, F., Gruenheid, S., Picard, V

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Gene therapy and NAD+ precursor (B3) supplementation treatments aimed at inducing NAD+ production have been successful at preventing further RGC and visual function loss, which suggest that NAD+ generation may be a key target for mitochondrial homeostasis and neuroprotection in RGCs (Cimaglia et al., 2020)

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Triglyceride hydrolysis in hepatocytes is impeded by the PNPLA3 gene, especially the I148M variation, which causes lipid retention in the liver