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Subsequent therapeutic studies demonstrate that the BCL-2i ABT-199 exhibits single-agent efficacy in all 3 BCL-2 overexpressing AI models whereas Enza and ABT-199 combination manifests synergistic potency in inhibiting the AR + BCL-2 + (i.e., double-positive) LNCaP-AI

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Supporting may also be the BPC 157-induced decrease of the inflammatory mediators, leukotriene B4 (LTB4), thromboxane B2 (TXB2), and myeloperoxidase (MPO), in both serum and inflamed tissue [40], largely implicated in heparin-induced platelet activation, the adhesion of leukocytes to the endothelium and concomitant increase in vascular permeability [41], prolongation of the bleeding time [42] while low-molecular-weight heparin enoxaparin causes systemic MPO activation [43]