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[ 2,3] Deficits in cholinergic function are a major feature of age-related declines in memory, with loss of cholinergic neurons (i.e., the nerve cells that make and release acetylcholine) and decreased ability to make acetylcholine contributing to the poorer memory and attention

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Polyunsaturated fatty acid (PUFA)-containing phosphatidylethanolamine (PE) is more susceptible to lipid peroxidation

These intricate EC-NO interactions highlight therapeutic opportunities for neurodegenerative and cardiovascular disorders through receptor-specific modulation of NOS activity (Park et al

It may have a role in in cell survival and the repair and regeneration of damaged tissue, but human clinical trials are limited