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In this highly heterogeneous context, the identification of recurrent biological features (i.e., biological endophenotypes) or recurrently affected pathways can be a key to understand the mechanisms underlying ASD

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26 Secondly, they found that MYC controlled GLS1 expression post-transcriptionally through microRNAs miR-23a and miR-23b in PC3, P493-6, human lymphoid CB33 and breast cancer MCF-7 cells

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