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The researchers concluded that defense mechanisms against reactive oxygen species are impaired in RA. To me, this sounds like a strong reason for anyone with RA to add active glutathione to their daily regimen

The mechanism involves an initial phase of acute hypoxia (ischemia) due to disrupted blood circulation, followed by a second phase of reperfusion injury when blood flow is restored, which exacerbates tissue damage (268)

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Consistent with this hypothesis are data demonstrating that restoring or bolstering physiological SOD1 structure and function is associated with higher neuronal densities in vulnerable populations, and longer survival times, in animal models of all three disorders.[ 151 , 154 , 182 , 209 ] We recently proposed that biochemical similarities between vulnerable neuronal populations in ALS and Parkinson's disease promote the evolution of a shared detrimental biomolecular cascade, within which misfolded SOD1 constitutes a key therapeutic target