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This approach accelerates the transition from bench to bedside, facilitates high-throughput screening and provides insights into drug mechanisms, supporting the development of personalized treatments by leveraging existing medications and providing hope for improved treatments for epilepsy patients

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SS-31 research is therefore most relevant when the study question involves mitochondrial dysfunction, impaired ATP output, oxidative stress, cardiomyopathy, skeletal-muscle fatigue, primary mitochondrial disease, or age-related mitochondrial decline