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This underscores the critical role of SLC25A39-mediated mitochondrial GSH import in maintaining cellular redox balance and overall mitochondrial function

Abstract Background: Iron overload cardiomyopathy (IOC) is a major co-morbidity of genetic hemochromatosis and secondary iron overload with limited therapeutic options

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Your liver purifies the toxin that enters your body

It has been shown that pharmacological inhibition or knockdown of a single mGSH transporter significantly sensitized neurons to oxidative and nitrosative stress (Wilkins et al., 2013)