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Mitochondrial dysfunction would appear to be the ideal candidate for a possible cause for ASD given the critical role mitochondria play in ATP production, calcium homeostasis, and synaptic plasticity within neurons (112, 113)

The dual approach produces additive and in research settings, often supra-additive reductions in food intake and body weight compared to either pathway alone, while semaglutide's glucose-dependent insulinotropic action and cagrilintide's glucagon suppression jointly support more stable postprandial glucose handling

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Comprehensive in vivo and in vitro studies demonstrated its effect in the prevention of neuronal loss and promotion of motor neuron survival, even after the onset of symptoms

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