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Mechanisms of myelin injury Multiple sclerosis lesions are characteristically perivascular and exhibit prominent demyelination and bystander axonal damage

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So far, our understanding of the dynamics of this process is a challenge that can be met with new tools of immunometabolomics

Patterns of v3 expression in primary and metastatic human breast cancer as shown by 18F-galacto-RGD PET

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This mechanism has been demonstrated in murine macrophages, where lipopolysaccharide-induced TNF- release upregulated CaSR and subsequently inhibited TNF- synthesis