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It produces a natural effect through the human bodys liver

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FDX1 is a small protein from the ferredoxin family and contains an Fe-S cluster ( 2+ and release Cu + into the mitochondrial matrix ( + can directly bind to Fe-S clusters, which in turn disrupt their synthesis and function ( Figure 3 ) ( Figure 3 4 Abnormal copper metabolism and liver disease The liver plays a central role in copper metabolism, including absorption, storage, transportation and excretion

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This relationship has been observed during seed dormancy removal (Oracz et al

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