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El-Zayat et al

Consistent with these findings, our current investigation revealed that the knockdown of SLC25A39 resulted in heightened ROS accumulation, suggesting that the reduction in mitochondrial GSH import mediated by SLC25A39 knockdown impairs the cell's ability to effectively eliminate or neutralize ROS generated from mitochondria, thereby leading to increased ROS levels in colorectal cancer (CRC)

10.2174/1381612811319120019 Curr

Supports endothelial cell proliferation and migration : VEGFR2 modulation is associated with increased fibroblast activity and early connective tissue regeneration in transected ligament models 4

doi: 10.1007/s10157-016-1288-2

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