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This phenomenon, characterized by heightened hydrogen peroxide levels in muscle cells due to diminished blood catalase, is postulated to potentiate insulin signaling through oxidation-sensitive tyrosine phosphatase inactivation, impairing insulin receptor dephosphorylation [84]

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Bhandari R, Yang H, Kosarek NN, Smith AE, Garlick JA, Hinchcliff M, et al

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