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In in vitro and in vivo studies, AC suppressed the vascular endothelial injury induced by complement noted by lowered adhesion molecules (ICAM-1, VCAM-1), and NF-B transcriptional activity, significantly reducing leukocyte infiltration, as well BALF protein content, and pulmonary MPO activity (Guo et al

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de Almeida BrasielPGDutra MedeirosJBarbosa Ferreira MachadoASchuchter FerreiraMGouveia PeluzioMCPotente Dutra LuquettiSC

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