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IGF-1 increases the potency of kainate-dependent currents in cerebellar granule neurons and modulates Ca 2+ , Cl , and K + channels by PI3K-dependent pathway, but not MAPK dependent pathway [73, 79]

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The membrane was then saturated for 1 h with TBS Odyssey Blocking Buffer (LI-COR) and incubated overnight with primary antibodies (Table S1)

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These findings have significant implications for the development and monitoring of NAD-targeted therapies, emphasizing the need for personalized approaches in metabolic interventions

Parker-Athill and Tan, 2010