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[16] Hexokinase I is the predominant isozyme in the brain

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One plausible explanation for these results is that the gpoA mutant is unable to effectively counteract the elevated ROS concentrations encountered within the intracellular environment of host cells, despite the partial compensatory activity of the TpxD and AhpD peroxidases (Fig

Alternatively, other inter-laboratory differences in culture conditions could be operative here, and possibly non-publication of negative findings (publication bias)

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