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Matrix metalloproteinases in the brain and bloodbrain barrier: versatile breakers and makers

The takeaway: strong human data on the anchor, solid pharmacology on the amplifier, and a genuine gap where the blend itself would need its own trial

The mechanism involves protecting neuronal mitochondria from oxidative stress while supporting neurotransmitter synthesis pathways

Finally, autophagy, the process of degrading misfolded proteins and damaged organelles, is required for cell survival but becomes aberrant under stress conditions

These inconsistencies point to the need for further research to better understand how factors like patient population, dosage, and underlying health conditions may influence LCs impact on BP