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Because astrocytic [Cl ] i is critical for regulating neuronal inhibition, a key question arises: are [Cl ] i shifts merely a byproduct of distinct neuronal activity patterns in different brain states, or do they actively shape neuronal excitability on a state-dependent basis

Supporting these observations, single-cell RNA sequencing and quantitative immunofluorescence analyses of control and IPD midbrain sections indicated an upregulation and activation of astroglial cells in the patient tissue (140)

Its a reliable model for studying growth pathways, making it valuable for both basic and applied research

PMID 17631101

Index markers of chronic fatigue syndrome with dysfunction of TCA and urea cycles

doi: 10.1038/srep39045 (2016)