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In other experimental AKI conditions, ferroptosis inhibitors have demonstrated protective effects against tubular damages in rhabdomyolysis-associated AKI, folic acid-induced kidney injury, and oxalate-induced kidney injury [30, 34, 35]

Because endogenous amylin has a relatively short half-life, scientists have explored ways to create modified analogs capable of maintaining biological activity for longer periods

FOXO4-DRI, designed to disrupt the FOXO4-p53 interaction, was tested on senescent TM3 Leydig cells

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Both peptides are given in the form of an injection

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