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The second approach relied on chronic continuous exposure to clinically relevant drug concentrations for 721 days

Various mechanisms include targeting protein aggregates (e.g., with ASOs, antibodies, or other methods), enhancing autophagy or exocytosis, or reducing apoptosis in response to ER stress

Glutathione S-transferases (GSTs) catalyze the attachment of GSH to antibiotics or their reactive byproducts, often leading to inactivation

Furthermore, dietary Met supplementation abolished oxidative stress and inflammation, selectively modulated the expression of multiple cognition-related genes and proteins, and increased flux via the transsulfuration pathway in the hippocampi of aging mice, with significant increase in H 2 S and glutathione production

The acute toxicology in animal models appears relatively benign, but animal toxicology does not establish human safety across chronic exposure

As discussed, no antidote has been introduced to alleviate the toxicity of AFs and current management scenarios of AFs complications are based on the removal of these mycotoxins in foods