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After examining the role of UA and GPX4 proteins in ferroptosis, we discovered that UA specifically binds to the GPX4 protein through molecular docking

Targeted inhibition of mitochondrial iron overload or ROS accumulation effectively inhibited oxidative stress-induced ferroptosis

It should also be noted that the extracellular signaling by IL-1 is inhibited by excessive ROS, but is permitted by ROS-dependent S-glutathionylation, which protects its functionality [94]

This is likely due to the inherent differences in the Australian population of patients diagnosed with HCC compared to the IMBRAVE050 cohort

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McGrath, Campbell, Parks, & Burton, 2000)