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It is present in normal human plasma at concentrations ranging from 0.1M to 1.4M and this can increase to over 10 times higher during oxidative stress in vivo (Zhang et al., 2007)

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Li (Jiaqi et al., 2023) found that ADE could induce mitochondrial dysfunction, evidenced by elevated levels of mitochondrial ROS release, depolarization of the mitochondrial membrane potential, and decreased mitochondrial ATP

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