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First, using germ-free animals, directional microbiota transplantation, and gene editing techniques to clarify the causal mechanisms by which protective genera such as Bifidobacterium regulate the hepcidin-FPN1 axis and key molecules of chondrocyte ferroptosis (GPX4, SLC7A11), thereby consolidating the logical chain of microbiota-iron metabolism-cell death. Second, leveraging longitudinal cohorts and multi-omics technologies to reveal the time-dependent evolution of the gut dysbiosis-iron overload-ferroptosis-intestinal dysfunction cycle and identify key windows for early intervention

Collectively, the existing evidence indicates that multiple oncogenic signaling pathways, such as c-Myc, Kras, and HIF-1, directly regulate key processes in glutamine metabolism, thereby driving the tumor dependence on glutamine

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Justin Laube, MD Medical Reviewer Justin Laube, MD, is a dual board-certified integrative and internal medicine physician, retreat leader, and healing educator with over 15 years of experience caring for patients w...