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(91) demonstrated that disrupting mitochondrial activity in cultured MIO-M1 human Mller cells results in decreased basal ATP turnover and maximal respiration

Those cells also showed aberrant levels of mitochondrial superoxide and lipid peroxidation, both hallmarks of the oxidative stress response, which strongly correlated with caspase-1 activity

The animal foundation Beneath the two human studies sits the preclinical work

[PubMed] [Google Scholar] 89.Katar S Glucose-6-Phosphate Dehydrogenase Deficiency and Kernicterus of South-East Anatolia: J Pediatr Hematol Oncol

LPS triggers inflammatory pathways in cardiomyocytes, leading to oxidative stress and apoptosis, which ultimately result in cardiac damage (171)