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Notably, percentages of TM-expressing classical monocytes were increased in patients with lower blast counts and in patients with ring sideroblasts (erythroblasts with mitochondrial iron accumulation) ( S100A8 mediator) and pre-mRNA splicing in MDS patients with SF3B1 -K700E mutations, resulting in the upregulation of pro-inflammatory signaling pathways ( SF3B1 mutations have been linked to over-activation of nuclear factor kappa B (NF-B) signaling and increased production of inflammatory cytokines (114)

10.1016/S0891-5849(97)00286-4

For instance, GSH can be depleted, through inhibition of its synthesis using an inhibitor of GCL (buthionine sulfoximine, BSO) (57) under conditions that maintain the intracellular cysteine pool intact,